Chapter 2458 [2458] This is the disease


    Zhu Xing''s mother recalled with difficulty what the attending doctor had said. It was too difficult for her, her brain was so tired that she was about to go on strike.


    The doctor can only help the family recall the important content rted to the child''s life word by word: "Zhu Xing''s heart has expanded to the size of a ball. In his case, the condition can only get worse and worse. In that case we have to consider giving him a heart transnt."


    Heart dtion, at first nce it sounds like the dted cardiomyopathy we talked about earlier. Dted cardiomyopathy is a type of cardiomyopathy.


    Cardiomyopathy is divided into primary and specific (secondary), and the ssification concept of primary and secondary is consistent with other diseases and will not be discussed.


    Dted cardiomyopathy is a primary, but there are many categories of primary cardiomyopathy far beyond dted cardiomyopathy. Zhu Xing''s heart dtion is a primary cardiomyopathy, but it is not the dted cardiomyopathy we talked about before, but a gic cardiomyopathy called congenital insufficiency of myocardialpaction.


    This disease is rtively rare.


    Why is it rare, because it is either very insidious and has been present in the patient''s body without developing the disease, or the patient''s condition is already very serious once it urs. This disease cannot be detected through routine physical examinations such as electrocardiogram or routine auscultation. To find out, need to rely on more sophisticated equipment, including echocardiography, maic resonance CT and other means. If the patient is asymptomatic, who will do these further examinations, there will be none, which makes it difficult for doctors to detect it at an early stage.


    What kind of disease is this disease? Since it is a gic disease, the root cause is the dominant or recessive inheritance of human pathogenic genes. It is manifested in many patients with family history, in which the patient''s heart has problems during embryonic development.


    Our heart does not develop coronary arteries in the first month of embryonic development. At this time, the mainponents of the myocardium are trabece and recesses, and the blood supply of the heart depends on the recesses. These structures belong to the non-densifiedyer of the ventricr wall pathologically. In normal embryonic development, in the fifth and sixth weeks, the non-densifiedyer will densify and grow out of the epicardium and endocardium, and the crypt willpress into the coronary system.


    Like in patients with nopaction of myocardium, myocardial densification fails at this time point in embryonic development, resulting in pathologically visible thickening of the nonpactedyer of the ventricr wall. In this way, the patient''s myocardium is always mainlyposed of muscle trabece and recesses, and the grooves and grooves are inteced like awork, which is like a spongy shape, so this disease is also called spongiform cardiomyopathy.


    Spongiform cardiomyopathy It can be an isted disease with only this symptom, or it can beplicated by other cardiac malformations. In pediatrics, isted myocardial nopaction is the main disease.


    Zhu Xing is a typical congenital isted myocardial ipaction. Not only that, his father died suddenly, and it is not ruled out that he died because of this disease.


    In summary, the most damning thing about this disease is that if it has a small lesion, the patient can have normal heart function for a long time, confusing the patient and the doctor. When it urs, the lesions are veryrge, and the patient will develop heart failure, and then it deteriorates into intractable heart failure and severe arrhythmia. Like Zhu Xing, the dted heart cannot be reversed. Like dted cardiomyopathy, the original heart surgery can only change the heart structure and cannot correct the diseased myocardium. The only way to save life is heart transntation.